Human PLGF enzyme-linked immunoassay kit

CAT: EH0288 Datasheet
Specification 96 Test
Sensitivity 0.83 pg/ml (50 μl)
Standard Curve Range 4.10~1000 pg/ml
Standard Curve Gradient 7 Points
Number of Incubations 2
Detectable sample Liquid phase sample of soluble substances. For example: serum, plasma, cell culture supernatant, tissue grinding liquid, etc.
Sample Volume 50 μl
Type Fully Ready-to-Use
Operation Duration 120min
pg/ml O.D. Average Corrected
0.00 0.0377 0.0413 0.0395
4.10 0.0677 0.0737 0.0707 0.0312
10.24 0.1165 0.1256 0.1211 0.0816
25.60 0.2566 0.2508 0.2537 0.2142
64.00 0.5458 0.5588 0.5523 0.5128
160.00 1.1780 1.1940 1.1860 1.1465
400.00 2.4310 2.4210 2.4260 2.3865
1000.00 3.9734 3.8360 3.9047 3.8652

Precision

Intra-assay Precision Inter-assay Precision
Sample Number S1 S2 S3 S1 S2 S3
22 22 22 6 6 6
Average(pg/ml) 17.7 97.2 294.9 17.4 92.3 284.1
Standard Deviation 0.8 3.7 11.0 0.8 4.2 10.2
Coefficient of Variation(%) 4.5 3.8 3.7 4.7 4.6 3.6

Intra-assay Precision (Precision within an assay) Three samples of known concentration were tested twenty-two times on one plate to assess intra-assay precision. Inter-assay Precision (Precision between assays) Three samples of known concentration were tested six times on one plate to assess intra-assay precision.

Spike Recovery

The spike recovery was evaluated by spiking 3 levels of human PLGF into health human serum sample. The un-spiked serum was used as blank in this experiment.
The recovery ranged from 81% to 109% with an overall mean recovery of 93%.

Sample Values

Sample Matrix Sample Evaluated Range (pg/ml) Detectable (%) Mean of Detectable (pg/ml)
serum307.82-29.54100.014.90

Serum/Plasma – Thirty samples from apparently healthy volunteers were evaluated for the presence of human PLGF in this assay. No medical histories were available for the donors. n.d. = non-detectable. Samples measured below the sensitivity are considered to be non-detectable.

Background: PLGF

PlGF (placenta growth factor) is secreted primarily by villous trophoblasts and decidual cells, with circulating levels increasing during pregnancy but attenuated in preeclampsia. PlGF signals through VEGF R1/Flt‑1 and Neuropilins, while VEGF binds both VEGF R1 and R2 but signals mainly through VEGF R2. PlGF reduces binding of VEGF to VEGF R1 and increases VEGF/VEGF R2‑mediated angiogenesis. It induces monocyte activation, migration, and production of inflammatory cytokines and VEGF. These activities facilitate wound and bone fracture healing and also contribute to inflammation in active sickle cell disease and atherosclerosis.

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